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Finally, emerging evidence indicates that chitosan-based carriers (l-serinemodified chitosan) can exploit kidney injury molecule-1 (KIM1), a phosphatidylserine receptor markedly upregulated on the apical membrane of injured proximal tubular epithelial cells [90], to achieve active, injury-selective drug delivery [91]

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The reduction of pro-inflammatory cytokines, improvement in intestinal barrier integrity, and potential modulation of gut microbiota composition represent mechanisms that could theoretically complement existing IBD therapies

If retinopathy is present, more frequent examinations are needed based on severity