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Mitochondrial protein hyperacetylation in the failing heart
Liver damage, inflammation, and enhanced tumorigenesis after persistent mTORC1 inhibition
first proposed ferroptosis in 2012 as a form of programmed iron-dependent cell death, exhibiting unique characteristics at morphological, chemical, and genetic levels compared to other forms of cell death, such as apoptosis, necrosis, and autophagy (Li and Huang, 2022)
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