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Finally, patient lifestyle factors, such as smoking and alcohol consumption, are also linked to the modulation of cancer risk by GLP-1RAs

Degradation of glucagon-like peptide-1 by human plasma in vitro yields an n-terminally truncated peptide that is a major endogenous metabolite in vivo

It has been reported histone lactylation, especially H3K18la, can promote gene transcription including numerous pro-fibrotic genes [17, 29, 32]

GLP-1 RA Addition to Background Sulfonylurea Therapy Similar to the discussion above about background insulin, it is prudent in many cases to hold or reduce the dose of background sulfonylurea therapy (an initial 50% dose reduction is reasonable in many patients) when starting a GLP-1 RA