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Opposing roles of p38 and JNK in a Drosophila model of TDP-43 proteinopathy reveal oxidative stress and innate immunity as pathogenic components of neurodegeneration
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M9a + sh EPAS1 cells upregulate HIF-1-BNIP3/NIX-mediated mitophagy and suppress mtDNA-mediated inflammation under hypoxia condition Hypoxia-induced mitophagy is typically mediated by BNIP3/NIX and regulated by HIF-1 [28], so we first excluded the involvement of PINK1/PARKIN-mediated mitophagy in Tibetan hypoxia adaptation (Supplementary Fig
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