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In addition, studies have reported increased expression of growth hormone receptors in tendon-derived fibroblasts following peptide exposure, a response that may amplify regenerative signaling during joint recovery processes (20)
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GHK-Cu acts at the extracellular matrix (ECM) level, modulating the balance between matrix metalloproteinases (MMPs) and their inhibitors (TIMPs) to regulate collagen turnover, and stimulating fibroblasts to produce collagen type I, collagen type III, and elastin
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