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Glucagon-like peptide-1 decreases endogenous amyloid-beta peptide (Abeta) levels and protects hippocampal neurons from death induced by Abeta and iron
By combining these components, Klow may support study designs that: Compare different peptide types within the same experimental framework
Pothos E, Rada P, Mark GP, Hoebel BG
The peptides half-life in vivo is short due to rapid metabolism, which is why repeated administration or formulation stabilization is needed in experimental settings
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