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Experimentally, when retinoic acid, the most active metabolite of vitamin A, is administered in noise-exposed mice, it protects the cochlea by reducing significantly the apoptosis of hair cells and consequently, inducing a faster recovery of the auditory thresholds when compared to untreated animals (Ahn et al., 2005, 2013
Furthermore, there was an increase in the microphage activation marker Iba-1 in the spinal cord, suggesting that phagocytosis occurred in response to myelin debris production [82]
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