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Similar to this, the transmembrane adhesion molecule glycoprotein basal cell adhesion molecule (Lutheran blood group) and its particular integrin-4-1 mediate the attachment of sickle cells to the endothelium [40]
Blood viscosity is increased in these autoimmune and inflammatory conditions, contributing to decreased blood flow
Benoist 2011, PMID 21719467, which studied Nle1-AngIV analogs rather than Dihexa itself)
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