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After being returned to their racks mice were checked for signs of pain and distress and their weight monitored daily
This phenotipic transition promotes the release of pro-inflammatory cytokines and activation of cerebral endothelial cells, further compromising BBB integrity ( Persistent neuroinflammation may also promote the leakage of pro-inflammatory cytokines into the systemic circulation, transforming a localised central nervous system response into a generalised systemic inflammatory state ( 2.3.2 Direct catecholamine-mediated myocardial overstimulation The autonomic and inflammatory consequences of neuroinflammation converge on the myocardium through a second, overlapping mechanism: direct catecholamine-mediated cardiac injury
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Altered endocrine, cytokine signaling and oxidative stress: a plausible reason for differential changes in testicular cells in diet-induced and genetically-inherited obesity in adult rats
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