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Zinc deficiency can prevent CYP enzymes from functioning properly (11), potentially leading to inefficient detoxification
GSDMD, Gasdermin D
A key extrinsic pathway component is the death receptor Fas (CD95), which, upon binding to Fas ligand (FasL/CD95L), recruits the adaptor protein FADD (Fas-associated death domain protein) and procaspase-8 to form the death-inducing signaling complex (DISC)
Finally, we discuss the possible impact of Nrf2 inhibitors on cancer therapy
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