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From a theoretical pharmacological standpoint, combining a peptide with putative lipolytic activity (AOD 9604) with a potent triple incretin receptor agonist (retatrutide) could potentially amplify metabolic effects but this is speculative and raises the possibility of compounding adverse effects with no safety data to guide dosing or monitoring

However, it is important to emphasise that whilst these mechanisms are biologically plausible, there is insufficient high-quality clinical evidence for glutathione supplementation as a treatment for fatty liver disease

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