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Understanding the pathophysiology of gastric cancer and developing new therapies for gastric cancer depends on more researches focusing on the potential contributors to oxidative stress and gastric carcinogenesis
We therefore hypothesized that the pronounced increase in lipid metabolism, together with EBV-driven induction of oxidative phosphorylation reactive oxidative species (ROS) (12, 28, 29), sensitizes newly infected B cells to ferroptosis
Rozman Grinberg, I
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