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Dabbaghi Varnousfaderani S, Musazadeh V, Ghalichi F, Kavyani Z, Razmjouei S, Faghfouri AH, et al
Indeed, Sp was able to restore cytosolic and mitochondrial redox balance in neuronal cell line SHSY-5Y subjected to 1-methyl-4-phenylpyridinium (MPP + ) treatment which is known to cause mitochondria dysfunction and oxidative stress through complex I inhibition 1 (Fig
As electrons are delocalized over both iron and sulfur ions in the cluster 10 , Fe-S clusters are highly suited for electron transfer, redox sensing, or catalysis
In kidney aging studies, it protected renal tubular epithelial cells, reduced renal inflammation, and improved renal function
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