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Tamoxifen is metabolized through oxidative reactions catalyzed by a series of cytochrome P450 to its more active derivatives 4-hydroxy-tamoxifen and 4-hydroxy-N-desmethyltamoxifen (endoxifen)
To further support a role for GLP-1R signaling in regulating food reward, recent fMRI studies in obese and obese T2DM patients have demonstrated that intravenous infusion of exenatide decreases the activation of brain regions involved in mediating food reward (van Bloemendaal et al., 2014)
Clinical trials showed no thyroid cancer signals, and the medication is contraindicated only in patients with personal or family history of medullary thyroid carcinoma or MEN 2 syndrome
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