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Its association with several other major downstream pathways contributing towards cancer proliferation and metastasis has made it to be explored more and find suitable targets against it
Mechanistically, it acts as a ghrelin mimetic, binding the growth hormone secretagogue receptor (GHS-R1a) the receptor for the endogenous peptide ghrelin, expressed in the pituitary and hypothalamus
supplementation may reduce severity in deficient individuals, with serum zinc testing required to guide appropriate intervention [11]
There is a paucity in the literature of papers addressing molecular mechanisms of new peptide polyagonists and extending these to preclinical and clinical data
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