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In addition, ferroptosis signals spread to adjacent cells, possibly through release of oxidized lipids through extracellular vesicles, leading to ferroptosis in adjacent cells (Riegman et al

Researchers have nominated several candidates for this role over the years, but none have been definitively crowned

Our work positions TET1 as a pivotal epigenetic hub governing ferroptosis surveillance, and provides a translatable strategy to overcome ferroptosis resistance in cancer, with AML as a paradigm

Tesaglitazar is a PPAR/ dual-agonist, which in phase II and III clinical trials improved glucose and lipid metabolism with greater efficacy relative to selective PPAR agonism 13,17,18,19,20,21