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GRK5 and GRK6 are more prominent in the CNS meaning the same agonist writes a different phospho-barcode in different tissue contexts
As agonist dissociation within endosomes is a determinant of post-endocytic targeting to recycling or degradative pathways 44 , these observations may explain the contrasting recycling patterns of exendin-phe1 vs
Fission and selective fusion govern mitochondrial segregation and elimination by autophagy
Here's what the actual research says, and where it falls apart
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