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Conclusions Collectively, NAC could ameliorate COPD-induced pulmonary fibrosis by promoting immune response and inhibiting EMT process via the VWF/p38 MAPK axis, therefore providing us with a potential therapeutic target for treating COPD
The prevalence of the 718TC and 718CC genotypes of the GPX4 gene was higher in the EHCS group than the EH-alone group (OR=2.12, 95%CI 1.423.16, P adj =0.0018)
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