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(G12D) NRAS and kinase-dead BRAF cooperate to drive naevogenesis and melanomagenesis
It may possibly reflect differences in the occupancy of the hydrophobic pocket in VP1, or at another site affecting stability, since the ternary complexes of PV3-SC8 GPP3+GSH and PV3-SC8 pleconaril+GSH demonstrate that the bound factors could act additively or synergistically
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