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Significant targets and the effects of PKGI in SMC are activation of myosin light chain phosphatase, inhibition of PLC, inhibition of the IP 3 receptor, inhibition of plasma membrane localized L-type Ca 2+ channels, and activation of plasma membrane localized calcium-activated K + channels (commonly called the big potassium, BK, channels)
The figure is biologically attractive and genetically coherent, but no primary-prevention trial has tested whether treating healthy adults to below 0.6 g/L produces better outcomes than current practice
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