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Specifically, phosphorylation of GR at Ser203 impedes nuclear translocation and reduces GR activity, whereas phosphorylation at Ser211 enhances nuclear translocation to augment GR signaling (326, 332)
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Combination research specifically explores whether the non-overlapping mechanisms produce additive or synergistic effects in tissue-repair model systems, a multi-mechanism approach that single-peptide pharmacology cannot address
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