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We have used whole-genome transcriptional profiling analysis and various quantitative phenotypic assays to identify gene expression and associated phenotypic differences in parental and GSTP1 knockdown cell lines
doi: 10.1111/j.1749-6632.2010.05875.x 33
The increase in m by low v-ATPase deficiency might also decrease vulnerability to other mitochondrial dysfunctions, as ATP6AP1 KO cells are insensitive to artificially lowering m by FCCP, providing a link to m -dependent OPA1 processing rescuing organelle architecture 75
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