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Under hypobaric hypoxia exposure conditions (7620 m, 8% O2, for 7 days), increasing H2S levels inhibited hypobaric hypoxia-induced apoptosis in hippocampal neurons and also significantly protected glial vascular homeostasis and key neurophysiological functions (such as cerebral blood flow, functional congestion, and spatial memory) (Kumar et al., 2016a)
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