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Following FOXO4 displacement, the released p53 undergoes nuclear export and mitochondrial translocation, activating the intrinsic apoptosis pathway via BAX upregulation and BCL-2 downregulation, culminating in caspase-3 cleavage and apoptotic cell death

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Preclinical reports describe involvement of VEGFR2 signalling and the nitric oxide system in rodent models of tendon and soft-tissue repair