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[79] showed that adenovirus-induced OGA overexpression and OGT knockdown failed to restore impaired Akt activation by insulin stimulation and prevent glucose-induced insulin resistance in 3T3L1 adipocytes, and in another study, treatment with the OGA inhibitor 1,2-dideoxy-2-propyl---glucopyranoso-[2,1, D]-2-thiazoline (NButGT) did not induce insulin resistance characterized by defective glucose uptake and Akt phosphorylation in 3T3-L1 adipocytes [80]

Similarly, Liebmann et al

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129 Stabilized -catenin translocates to the nucleus and partners with TCF/LEF transcription factors to activate oncogenic targets, including MYC, Cyclin D1, LGR5, CD44, and ABCB1, genes that regulate proliferation, drug efflux, and stemness