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Given the extensive evidence linking redox dysfunction to ALS, it is not surprising that several redox-active molecules have been trialed as potential therapeutic agents
A recent report described that adenoviral overexpression of dominant negative type c-Jun N-terminal kinase-1 preserved PDX-1 DNA-binding activity in face of oxidative stress and protected insulin gene expression and secretion [55]
Epithalon injected to rats exposed to any photoregimen also significantly inhibited the development of spontaneous tumors, primarily testicular leydigomas and leukemias
The curious case of the tumour virus: 50 years of Burkitts lymphoma
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