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Our data help explain the experimental findings related to how the human body absorbs different forms of vitamin B12 during the treatment of vitamin B12 deficiency [110]
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These inflammatory mediators perpetuate systemic symptoms and drive further mitochondrial damage
The activity of recombinant TcGSTm02 could be inhibited by cyflumetofen and the enzyme catalyzed the conjugation of GSH to cyflumetofen [113]
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