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The Central Role of Enzymes as Biological Catalysts
Interestingly, the arrestin activity of the mutant GLP-1R showed a similar profile, but at lower levels, as those of the G protein activity when the same truncated peptides were used (Figure 5b)
25,26,27 In contrast, mutations in POLE/POLD1 enhance immunogenicity through hypermutation, 28 while excessive copy number alterations often drive immunosuppressive phenotypes and resistance
These effects usually fade with consistent use
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