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Effect of very high-dose thymidine infusions on leukemia and lymphoma patients
Given the robustness of this mitochondrial response, the multifaceted nature of PGC1 regulation in cells and a modest rescue at best by mTORC1 inhibition, we suspect that there is a degree of functional redundancy built into this metabolic plasticity system that can contribute to such an adaptive response, and complete inhibition of this system will prove difficult
The apparent targeting therefore arises primarily from localized delivery via the microneedle patch rather than active molecular recognition
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