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In pathological conditions, such as sepsis or hemolytic crisis, Hx is partially recycled based on non-human primate studies, while Hp is eventually degraded in lysosomes (Smith and McCulloh, 2015)

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Panel B of Figure 5 shows that the fluctuations in methionine input cause very large fluctuations in SAM but only modest fluctuations in intracellular methionine concentration, as suggested by the early methionine loading experiments of Finkelstein and co-workers [53, 54]