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The loss of oestrogen receptors in mutated cells, or an altered binding site of topoisomerase, resulting in reduced sensitivity to specific anti-cancer drugs, has already been reported [85, 86]
and 3-numerous confluent changes subcategorized into: (a) the hemorrhagic zone, (b) edema, (c) the loss of neurons, (d) vacuoles, and (e) the loss of spinal column tracts [3]
It has developed multiple proprietary technology platforms, including biased agonist discovery platform, long acting and oral peptide delivery platforms, and has identified a series of drug candidates based on these core technology platforms
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