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In particular, morphological hallmarks of eryptosis, like PS externalization, cell shrinkage, and membrane blebbing, are orchestrated by Ca 2+ influx [191]
Acetoacetate can be exported out of the mitochondria for conversion to acetoacetyl-CoA by a cytoplasmic acetoacetyl-CoA synthetase encoded by the AACS gene
prothymosin alpha (ProT, a nuclear protein involved in cellular proliferation and protection against apoptosis [192], whose interaction with KEAP1 has been found increased after cell treatment with the OS inducer diethyl maleate (DEM) [193, 194]
A pro-inflammatory state is created within the body through the stimulation of cytokines (pro-inflammatory molecules)
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