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As NAc is also an important brain region associated with GLP-1-induced feeding suppression, further electrophysiological studies are necessary to explore the contribution of glutamatergic neurotransmission to endogenously released GLP-1-induced suppression of high-fat food intake in the VTA
PMID: 25995620
Because no human studies exist, the impact on fat loss, SIRT1 activity, or mitochondrial repair in people remains unknown
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