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Previous study, suggests that DON oral dose induced anorexic POMC via NF-B activation by increasing the levels of proinflammatory cytokines in the hypothalamus and anorexic CCK production via increasing intestinal TRPA1 expression (84)
This extended duration of action made exendin-4 a promising candidate for therapeutic development
Obviously, further study is needed to accept or refute this proposition, and investigations are currently underway in our laboratory
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