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Increased TMAO levels in addition to endothelial dysfunction, vascular aging, and oxidative stresses have been shown to cause thrombotic events and platelet excitability in humans, which in turn induce atherosclerosis [69, 72]
The authors acknowledge the Department of Pharmaceuticals, Government of India, New Delhi for financial support under 4244 scheme for promotion of Research and Innovation in Pharma Med-Tech (PRIP)
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