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[5] [9] For individuals engaging in regular physical activity, these appetite-suppressing effects can present unique challenges
Finally, behavioral assessments were limited to specific domains and the observed biochemical changes cannot be interpreted as evidence of global neuroprotection, since behavioral deficits persisted and some molecular markers were negatively affected by treatment
Hyperglycemic conditions and the resultant metabolic disorder primarily disrupt autophagy by blocking autophagic flux and compromising lysosomal function, ultimately leading to stagnation of the autophagy pathway (146, 147)
PMID 12654482
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