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Thus, non-reactive substrate analogues are very weak competitive inhibitors of ScGrx7
Nasogastric aspiration at induction revealed unexpectedly large gastric residual volumes, supporting delayed gastric emptying as a plausible mechanistic explanation
In cancer, however, DDR exerts a paradoxical influence: while repair deficiencies promote mutagenesis and tumorigenesis, restoration or hyperactivation of DDR pathways in established tumors fosters resistance by enabling efficient repair of therapy-induced DNA damage
Hinojosa said
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