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IGF-1 LR3s markedly reduced IGFBP affinity allows researchers to study sustained, IGFBP-independent IGF-1R signalling in a way that is not practically achievable with native IGF-1
Though the results of these studies require replication and extension to human subjects, they provide a plausible physiological pathway through which enteric glia act as stress transducers and induce depression via alterations in gut-to-brain signaling
Warnings: Risk of Methemoglobinemia: Monitor patients for signs, especially those with risk factors such as G6PD deficiency
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