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Animal studies and early human research suggest these peptides may speed tendon-to-bone healing, reduce inflammatory cell buildup, and support better pain and function
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Classical exhaustion is typically driven by prolonged antigenic stimulation and is characterized by progressive loss of effector function, sustained expression of multiple inhibitory receptors (including programmed cell death protein 1, PD-1), andcriticallya stable exhaustion-associated epigenetic landscape that constrains reinvigoration even after checkpoint blockade ( A key unresolved question is whether perioperative immune dysfunction engages the core transcriptional circuitry of exhaustion
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