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Animal studies across multiple research groups show accelerated tendon, ligament, muscle, and gut tissue healing, operating through VEGFR2 angiogenesis pathways, the Akt-eNOS nitric-oxide axis, and ERK1/2 signaling
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Further analyses of differentially expressed genes between GLP-1MK-801 and semaglutide treatment groups support this notion by showing enrichment of functional terms pertaining to glutamatergic signalling and synaptic plasticity among the most upregulated genes
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