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Further animal studies are needed in SCI models to determine if the overexpression of ubiquitin or Uch-L1 can lead to resistance to neural injury or promote neuroregeneration or neuroplasticity with benefits to functional recovery
This finding was consistent with the marked reduction in liver degeneration observed in the combination therapy group at the same time point (Fig
MOTS-c A mitochondrial-derived peptide investigated for metabolic and energy-regulation questions that complement tri-agonist work
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