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The mechanistic trigger for loss of immune self-tolerance (which induces melanocytes apoptosis) is dysfunctional mitochondria resident in epidermal melanocytes ( + T cells (Figure 2
By upregulating VEGFR2 signaling, the compound promotes the formation of new blood vessels in damaged tissue a process called angiogenesis
It has been investigated for potential effects on skin pigmentation, based on proposed mechanisms including influence on melanin synthesis pathways
For example, assume the manager at Bullard Company will receive a bonus for reaching a certain profit target but expects to be $15,000 short of the target
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