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In preclinical models, BPC-157 has been associated with: Reduced inflammatory cytokine expression Improved transition from inflammatory phase to proliferative phase This creates an environment where fibroblasts can function more efficiently

This bioavailability gap means oral dosing requires higher amounts to achieve equivalent systemic exposure

Considering the short half-life nature of peptides, of which BPC-157 is an example, it can initially be that the agent is generally safe

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