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With GLP-1 treatment, a cholesterol-induced increase in the protein level of PAR was decreased in INS-1 cells compared with that in a single cholesterol stimulation group (Figure 3A)

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Ruchira Glazer, Global Head of Cardiovascular, Renal and Metabolic Development, added an unexpected twist: GLP-1s might actually help Novartis by boosting patient engagement with cardiovascular risk-factor modification, creating a more receptive market when its drugs arrive

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