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In MCF-7 cells, it induced apoptosis and blocked cell cycle progression, hence mediating cell death ( in vitro IC 50 concentrations may not directly reflect clinically achievable plasma or tissue levels, and translational interpretation requires pharmacokinetic validation

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have been funded by Estonian Research Councils personal research funding start-up grant no

On the other hand, increased residence in the stomach leads to a higher degree of dissolved drug available for absorption immediately after entering the small intestine, resulting in increased PK exposure and delayed time to maximum concentration (T max ) for Class II drugs