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The elevated ROS production in gastric cancer can initiate genotoxic consequences, contributing to genetic instability, DNA damage, metabolic adaptation, drug resistance and occasional cell death
In addition, there are also challenges related to the differences between semaglutide and trizepatide
This makes it relevant to laboratory investigations involving receptor cross-talk, metabolic adaptation, glucose-associated signaling, appetite-associated pathway biology, energy expenditure models, substrate utilization, hepatic metabolic signaling, and adipose-associated response systems
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