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This neuronal circuit driven by GLP-1 released within the OB could thus be involved in the physiological control of insulin secretion both in the pre-prandial and post-prandial states
clEDS is caused by homozygous or compound heterozygous pathogenic variants in the TNXB gene, which leads to nonsense-mediated mRNA decay of TNXB and result in complete absence of TN-X or in misfolding of the protein8
It is widely believed that delays in diagnosis and treatment significantly contribute to increased incidences of perforated appendicitis, which result in increased patient morbidity
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