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Longer duration of therapy provided to poorly controlled subjects could result in larger changes in Hb A 1c as well as more substantial changes in neuropathic symptoms and objective assessment of small nerve fiber density and function

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Conclusion and Future perspectives Since the first description of LHON in 1871, 19 and the subsequent discoveries of the roles of mtDNA and nDNA in mitochondrial genetic disorders in 1988 32,33 and 1995, 37 the field has transitioned from the pre-molecular era to the molecular era

No significant changes in lab tests (e.g., blood counts, liver/kidney function) have been observed