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Long-term inhibition of dipeptidyl peptidase IV improves glucose tolerance and preserves islet function in mice
Ryan et al
Class effects, which are directly related to the mechanism of action, are common to all DPP4i
Interestingly, the neural circuits responsible for the metabolic effects of GLP-1R agonism may be distinct from those mediating nausea or malaise (Huang et al., 2024), suggesting that future therapies could be tailored to preserve metabolic benefits while minimizing gastrointestinal side effects
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