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For each compartment, we first characterize single-layer signatures: transcriptomic markers of oocyte developmental competence, metabolic profiles reflecting follicular health, immune and decidualization programs defining endometrial receptivity, and embryo secretomic footprints

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71 In HPP, deficiency or dysfunction of TNSALP disrupts the metabolism of PLP leading to alteration of both extracellular and intracellular PLP levels

Since the cellular synthesis of glutathione is ATP-dependent, decreased ETC activity also undermines glutathione synthesis, further exacerbating oxidative stress in a vicious cycle [8]